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CRISPR/Cas9-engineered Gad1 elimination in rats leads to complex behavioral changes: Implications for schizophrenia

In the APP/PS1 mouse model of AD, TUDCA was able to restrain microglial and astrocyte activation and proinflammatory polarization and reduce A generation, thus improving the rescue of cognitive deficits [229]

Our results indicate that targeting MAOA may be a potential useful addition to combination therapy in treating different types of NE tumors

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