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Energy metabolism and glutamate-glutamine cycle in the brain: A stoichiometric modeling perspective

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In the context of energy perception disorders induced by SUA and mitochondrial dysfunction, when energy perception disorders and mitochondrial damage occur, abnormal energy metabolism and the resultant generation of reactive oxygen species activate a series of stress-sensitive signaling pathways, including the c-Jun N-terminal kinase, protein kinase C, and IKK /NF-B pathways (52, 53)

PMID: 160 CaoYLiYHeCYanFLiJRXuHZet al