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As erythrocytes from patients with inherited enzyme deficiencies (e.g., glucose-6-phosphate dehydrogenase-deficient cells) are more susceptible to different inducers of eryptosis, it seems reasonable to mention RBC enzyme deficiency-dependent erythrocyte death [80]

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As a cytoprotective agent, mediating Roberts cytoprotection, it maintains endothelium integrity and has a particular angiomodulatory effect (Sikiric et al., 1993