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It is noteworthy that gene-nutrient interactions, especially between the C677T mutation of the MTHFR gene and folate status and perhaps between heterozygosity for CBS defect and folate status (Lentz et al, 2000), could be involved in the onset of hyperhomocysteinemia

If you have genetic predispositions affecting nutrient pathways (detectable through tests like PlexusDx's Precision Peptide Genetic Test), this information should inform your provider's recommendation, dosing strategy, and monitoring plan

Closing the loop on the GABA shunt in plants: are GABA metabolism and signaling entwined