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Since the two peptides have complementary mechanistic profiles, theyre particularly useful for investigating multi-pathway repair

A deeper understanding of mitochondrial metabolic reprogramming in CRC, particularly its involvement in therapeutic resistance, metastatic adaptation, and metabolic vulnerabilities [14], is essential for the development of targeted interventions and the advancement of precision oncology

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349 Similarly, in tumor-bearing mice infected with Plasmodium yoelii , malaria infection suppressed major pathways that recruited Tregs to the TME by releasing exosome-like vesicles, resulting in the downregulation of cytokines and growth factors involved in Treg and MDSC recruitment