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Description

Niacin and selenium attenuate sepsis-induced lung injury by up-regulating nuclear factor erythroid 2-related factor 2 signaling

excessive ROS caused reduced m and ATP levels, and impaired mitochondrial function with altered mitochondrial dynamics, exacerbating inflammatory response and promoting COPD progression ( Mitochondrial quality control is a regulatory mechanism for the bioenergetic changes that occur in response to mitochondrial dysfunction (Springer and Macleod, 2016)