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Results c-Met is overexpressed and associated with PNI in pancreatic cancer To analyze the relationship between c-Met and PNI in pancreatic cancer, we performed immunohistochemical staining to examine c-Met expression in human pancreatic cancer tissues with or without PNI

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The peptide inhibited hyperglycemia-induced epithelial-mesenchymal transition (EMT) and downregulated fibrosis-related genes, preventing the subretinal fibrosis that represents end-stage proliferative diabetic retinopathy