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In particular, glial-neuronal communication through NMDA receptors is an essential process for proper dendritic morphogenesis and establishment of synaptic connections (Rabacchi et al., 1992

Studies in RGCs, specifically, have shown that exposure to extended periods of hypoxia (achieved through vascular hypoperfusion, evoked hypoxic states, or hypoxia-mimetic agents) induced ROS and oxidative stress elevation, upregulating pathways to RGC apoptosis (Tezel, 2006