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Yang et al., 2019), disturbed action potentials (Yang et al., 2019), as well as impaired neurite extension and increased mitochondrial biogenesis as a compensatory strategy to counteract the defective phenotype induced by LHON mutations (Wu et al., 2018)

The primary mechanisms can be summarized into the following three categories: 1

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Although a number of creative and successful approaches in animal models have been employed to deliver growth factors like GDNF (Wang et al., 2011) to the brain, these methodologies are technically complex and prohibitively difficult to bring to practice with large numbers of patients