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It is characterized by iron-dependent lipid peroxide damage in mitochondria and a lack of glutathione peroxidase 4 (GPX4) and is distinct from apoptosis, autophagy, and necrosis (Dixon et al., 2012) (Figures 1, 2)

Increased T H 17 signaling contributes to cystic fibrosis lung pathology, and cystic fibrosis sputa have elevated amounts of IL-17, further enhancing neutrophil recruitment in the cystic fibrosis lung 108, 109

Kuller has authored and co-authored more than 600 papers

Activation of CB2 receptors reduces inflammatory signaling by decreasing iNOS expression in central immune cells such as microglia and astrocytes (Gmez-Glvez et al