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Abstract Oxidative stress, arising from an imbalance between reactive oxygen species (ROS) production and antioxidant defenses, is increasingly recognized as a pivotal driver of cancer initiation and progression

This leads to IR, oxidative stress and inflammation through the release of inflammatory cytokinin promoting fat accumulation and tissue fibrosis [33, 35]

Alternatively, the more supported hypothesis is that there could be hyper-responsivity of the cholinergic receptors at a hypothalamic level, causing a greater decrease in somatostatin tone, and elevated GH release from the anterior pituitary gland (153)

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