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Alterations in the normal function of excitatory amino acid carrier 1 affect cysteine transport and GSH synthesis impairing zinc homeostasis (the thiol group of GSH can function as a principal Zn 2+ chelator for the maintenance of Zn 2+ homeostasis in neurons) and oxidative stress, enhancing susceptibility to ischemia-induced neuronal cell death in the hippocampus (162164)

In this paradigm, aquaporin 8 silencing, which is specifically expressed in IMM, enhances mitochondrial ROS generation and results in mitochondrial depolarization and cell death (Marchissio et al., 2012)

This reactive stroma substantially modifies the mechanical and biochemical properties of the TME, creating a favorable condition that supports tumor growth and immune evasion

Exposure to reactive oxygen or nitrogen species is causatively linked to the disease pathologies associated with redox imbalance