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UCB exhibits a rapid increase in ER stress characteristics, including Ca2+ release, calpain, activation of JNK1/2, and the production of ROS, mitochondrial dysfunction, apoptosis, and necrosis [46]

It is not clear how ROS contributes to the selection of a particular cell fate in erythrocytes, which might be an interesting topic for further research, especially given that sources of ROS differ in erythrocytes as compared with nucleated cells [54]