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While renal urate transporters have been extensively characterized over decades, their intestinal counterparts represent an emerging and highly dynamic research focus with substantial potential for managing hyperuricemia, offering a substitute excretory route (So and Thorens, 2010

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High levels of Ca 2+ /calmodulin promote CaM-KII binding to GluN2B-NMDARs, facilitating LTP, whereas dissociation is linked to LTD (Bayer & Giese, 2025)