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Availability: InStock
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The underlying mechanisms involve maladaptive auditory-limbic network connectivity and dysregulation of neuro-immune interactions, where astrocytes play a significant role in both neural responses to inflammation and the plasticity regulation implicated in tinnitus pathophysiology (Perin and Pizzala, 2024)

We have also recently reported that GRK4 increases the phosphorylation of angiotensin II type 2 receptor (AT 2 R) which impairs renal AT 2 R-mediated diuresis and natriuresis, which participate in the GRK4-mediated regulation of blood pressure [93].In addition to dopamine and Ang II receptors, GRK4 also regulates other receptors such as the endothelin type B receptor (ETBR) and AdipoR1