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Funding SD was supported by grants from the Wings for Life Spinal Cord Research Foundation (WFL, Salzburg, Austria

Conversely, cartilage-specific overexpression of GPX4 via transgenic technology, or restoration of its activity using small molecule drugs such as certain natural compounds or ferroptosis inhibitors, can significantly delay cartilage degeneration, improve subchondral bone sclerosis and osteophyte formation, and alleviate synovitis in model animals (Xiao et al., 2024), which directly confirms the protective role of GPX4 in OA progression from a gain-of-function perspective

This cascade leads to the activation of neutrophils and formation of NETs, which are important pathological factors involved in the acute inflammatory response, likely contributing to the organ damage seen in patients supported on ECMO