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By using publicly available RIP-Seq, RNA-seq and PAR-CLIP-Seq transcriptomic data, we showed, for the first time, that HuR binds to GLS mRNA at intron 14 (the intron before exon 15, which is the site of splicing that generates the KGA isoform instead of the GAC isoform) and at the 3-UTRs of GACs and KGA

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Androgen receptor inhibition suppresses anti-tumor neutrophil response against bone metastatic prostate cancer via regulation of TRI expression

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