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doi: 10.1016/0021-9150(79)90180-1 207 RastopchinI

Moreover, we also evaluated the downstream signaling of IGF1R and VEGFR2 in HUVECs cultured in CM of SKOV3shCtrl or shELF3 cells exposed to hypoxia or normoxia, and we did not observe increased expression and secretion of IGF1 under hypoxia, where ELF3 was silenced (Fig 5A)