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In addition, dhSph metabolism forms dihydrosphingosine-1-phosphate (dhS1P) under the action of sphingosine kinase, which can increase collagen synthesis in fibroblasts leading to fibrosis through dhS1P JAK/STAT-TIMP1 signaling (50, 51)

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Finally, other activation mechanisms contribute to the activation of the NLRP3 inflammasome, further aiding in the development of PD models in animals (Huang et al., 2024