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When the central repressors of ferroptosis, such as glutathione (GSH) and lipid enzyme glutathione peroxidase 4 (GPX4) [103], which participate in peroxide reduction and reduce product toxicity, show insufficient activity, the lipid radicals formed cannot be cleared and continue to generate new oxidative products via chain reaction

Intraputaminal AADC gene therapy (VY-AADC01) for advanced Parkinsons disease: interim results of a phase 1b trial

To interrogate further the functional role that this DP mutant could be playing in the regulation of Ect2, we engineered the same point mutation that results in the truncated form of DP in the Carvajal syndrome patient into the DPII-GFP construct and inserted it into our adenoviral vector (Fig 6B)

[10] NAD+ is not licensed as a medicine by the Medicines and Healthcare products Regulatory Agency (MHRA) for specific therapeutic indications