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WARNING: For research purposes only

Blockade of Eotaxin/Oncostatin M not only prevented hypoxic breast tumor cells from recruiting and polarizing macrophages towards the M2-like phenotype and hindered cancer progression in 4T1 breast cancer model but also improved the efficacy of antiangiogenic Bevacizumab, suggesting these two cytokines as novel targets for devising effective anticancer therapy (293)

The expression that comes up again and again in the US T1 community is: "We're hostages"

Effects on Fibroblasts and Extracellular Matrix AHK-Cu primarily affects fibroblasts, cells responsible for maintaining and growing the extracellular matrix (ECM)[1]