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These products are reversible by sulfiredoxins, but further oxidation by H 2 O 2 irreversibly yields sulfonates, which causes protein function loss, a process typically observed during oxidative distress

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Notably, this formulation also sensitised adriamycin-resistant CRC cells to 5-FU therapy by downregulating P-gp expression, a target of the NF-B signalling pathway [186]

In addition, erastin or RSL3, which are classical ferroptosis activators, can cause abundant intracellular iron ion accumulation and thus inhibit the intracellular antioxidant system and then increase damage caused by oxidation by generating ROS through the Fenton reaction