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Relevance of SIRT1-NF-kappaB axis as therapeutic target to ameliorate inflammation in liver disease

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Additionally, the pan-caspase inhibitor benzyloxycarbonyl-Val-Ala-Asp-fluoromethylketone (Z-VAD-FMK), the potent blood-brain barrier-penetrant necroptosis inhibitor necrostatin-1 (Nec1), and the selective ferroptosis inhibitor ferrostatin had no impact on the total number of viable cells and cytotoxicity (Supplementary Fig

In parallel, NF-B acts as the master switch for pro-inflammatory gene expression, and its hyperactivation drives the production of inflammatory mediators such as TNF- and IL-1[49-53]