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Obesity is an autonomous risk factor for cardiovascular diseases, including coronary artery disease, heart failure, and atrial fibrillation [115]

Indeed, this aspect has also been confirmed in different works where the increased ROS levels is related to the reduction of antioxidant enzymes following MNPs exposure [63,66]

Most conjugated products of GSTs are cytotoxic and therefore must be eliminated, glutathione S-conjugated products are exported from cells (in particular from liver cells where cytotoxins are concentrated) via a membrane ATP-dependent pump known as the glutathione S-conjugate export pump [4]

Intriguingly, PIK3C3/VPS34 lactylation augments its interaction with Beclin1, Atg14L, and UVRAG, increasing its lipid kinase activity and promoting macroautophagy/autophagy and endolysosomal degradation