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Using the framework of iron as a catalyst for lipid peroxidation, along with evidence of lipid peroxidation and decreased glutathione (the substrate for the ferroptosis checkpoint inhibitor, glutathione peroxidase 4, GPX4), ferroptosis has been implicated as a pathological contributor promoting neuronal loss [31,32]

Deletion of either exon 3 or exon 4 occur both in-frame and result in a non-functional protein lacking either the second zinc cluster or the hinge region and the NH 2 -terminal part of the ligand-binding domain [see Figure 7 for genomic organization of the androgen receptor gene]

The KPV peptide is among the most exciting peptide molecules yet to enter the spotlight