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Description

Previous studies suggested that ferroptosis is executed by peroxidizing PUFA-chains-containing phospholipids (PL), especially phosphatidylethanolamines (PE) and phosphatidylcholines (PC) [44], while the lysophospholipids (LP) increase upon ferroptosis inducer treatments as the product of enzymatic cleavage of the oxidized PUFA tail [45, 46]

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Specifically, the PTEN-induced kinase 1 (PINK1)/Parkin pathway is a classic mechanism that activates mitophagy by mediating the recognition of damaged mitochondria

Comprehensive analysis supports the fundamental difference between senescence and eryptosis, evidenced primarily by much faster clearance of eryptotic cells compared to senescent erythrocytes [78]