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Gut microbe-derived metabolite trimethylamine n-oxide activates the cardiac autonomic nervous system and facilitates ischemia-induced ventricular arrhythmia via two different pathways

Pharmacological treatments have proven effective but have not substantially improved the functional outcome for the majority of people with SCZ, as they are not effective on dimensions which account for most of the functional impairment of these people ( Both dimensional and categorical approaches have been adopted to reduce the heterogeneity of the syndrome

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