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Its effect was associated with a signicant de- crease in oxidative stress parameters and likely maintained NO system function, providing that BPC 157 interacted with eNOS and COX2 gene expression, in a particular way and counteracted the noxious effect of the NOS-blocker, L-NAME

Low Zn levels are associated with activation of Caspase-3 and downregulation of the ERK pathway, reduced Ki67-positive cells, increased TUNEL-labeled cells in the subgranular zone (SGZ), arrest of the cell cycle in the G0 G1 phase, promotion of apoptosis in neurons, and modification of cell signals that control the pro-survival and pro-apoptotic gene expression, such as NF- and p53 (189)

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