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Metabolic and alcohol-associated liver disease (MetALD): a representation of duality

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Its mechanism relies on spatiotemporal coordination among epidermal cells, immune cells, and fibroblasts through extracellular matrix (ECM) remodeling and signaling factor cascades, involving multi-dimensional molecular interaction networks such as gene expression regulation, metabolic reprogramming, and microenvironmental homeostasis

Compounded formulations use different excipients and may not have the same stabilization advantages