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all of which are key proteins involved in inhibiting ferroptosis, while idebenone pretreatment upregulated their expression by 1.44-fold, 1.51-fold, and 3.10-fold, respectively [1]

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This progressive oxidative burden is widely believed to represent a critical mechanistic bridge linking normal aging to the onset and progression of AD [4, 5, 7, 9, 10]

Here, we provide insights from a short-term experiment on the complex interactions between predicted scenarios, in which climate change stressors influenced estuarine mussel responses when associated with a mixture of metals in sediments