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Caspase-1 inhibitors (e.g., Ac-YVAD-CMK, VX-765) can alleviate pulmonary vascular remodeling, reduce right ventricular systolic pressure in MCT-induced PH rats, and significantly reduce the level of pulmonary fibrosis [134]

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By contrast, heterodimerization with AP-1 family proteins, such as c-Jun, redirects their binding toward AP-1 sites (74,78,92-94)

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