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This review integrates preclinical research related to the effects of both stress and antidepressant treatment on synaptic plasticity, and proposes normalization of synaptic function as a final common pathway for depression treatment

3 and Table 6)

has received funding from the NIH (R01 AI-103173)

3T3-L1 preadipocytes respond to IGF-1 LR3 through both IGF-1R and insulin receptor hybrid receptors, making them relevant for adipogenesis and metabolic signaling studies