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Gut-liver axis: further immunological connections Given the constant exposure of the intestinal epithelium to microbial antigens and its direct drainage into the portal circulation, immune surveillance at this interface is critical for preventing inappropriate immune activation in the liver [38]

Presynaptic mitochondrial calcium sequestration influences transmission at mammalian central synapses

In contrast to IL-10, tumor necrosis factor induced protein 3 (TNFAIP3), also known as an anti-inflammatory protein (43), was already downregulated at 24 h and reached again the initial level at 14 d pi

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