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The efferocytosis dilemma: how neutrophil extracellular traps and PI3K/Rac1 complicate diabetic wound healing

Iron overload enhances oxidative stress and the NACHT, LRR, and PYD domains-containing protein 3 (NLRP3) inflammasome signaling, triggering the evolution of several inflammatory mediators, resulting in a cascade of inflammatory responses and renal dysfunction in iron-overloaded rats (Chaudhary et al., 2018)

In addition, the mice also showed the phenotypes of increased mitochondrial oxidative stress, histopathological changes, and abnormal mitochondrial ultrastructure in cardiomyocytes

Furthermore, Bax alters mitochondrial outer membrane permeabilization antagonizing the function of Bcl-2, leading to the release of bioactive substances such as Cyt-C from the intermembrane space into the cytosol (59)