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The development of technologies such as CRISPRCas9, which is based on targeted DNA editing, has facilitated rapid progress in gene therapy, and these technologies have also shown therapeutic effects in mice with fragile X syndrome

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The ifd-1() and ifd-2() mutants exhibited WT morphology (Supplementary Fig

We model the reaction as linear and reversible because covalent binding of NAPQI gradually declines after eight hours[28, 29]