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Restricting -synuclein transport into mitochondria by inhibition of -synucleinVDAC complexation as a potential therapeutic target for parkinsons disease treatment

*Correspondence: Ning Shen, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

The name reflects the three amino acids that form the peptide structure: Glycine Histidine Lysine Although GHK itself is biologically active, its most studied and therapeutically relevant form is GHK-Cu, which is formed when the peptide binds with a copper ion

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