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Successful use of infliximab in macrophage activation syndrome with severe CNS involvement

A novel ferroptosis-related 12-gene signature predicts clinical prognosis and reveals immune relevancy in clear cell renal cell carcinoma

After a spinal cord injury, the nerve pathways that normally restrain excessive muscle activity may be damaged or interrupted

there is the potential for: Ischemic injury due to potent vasoconstrictor action and tissue hypoxia Mild bradycardia, probably as a reflex result of a rise in blood pressure Arrhythmias Anxiety Transient headache Respiratory difficulty Pharmacokinetics: When released from nerve terminals, its actions are primarily (75%) terminated by neuronal reuptake Diffusion and degredation by extracellualr COMT contribute to a lesser extent