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Several potential disease mechanisms have been identified, including immune abnormalities, inflammatory activation, mitochondrial alterations, endothelial and muscular disturbances, cardiovascular anomalies, and dysfunction of the peripheral and central nervous systems

A recent study provided insights that even 23 weeks of cuprizone ingestion is sufficient to induce glial responses and de- and remyelination pattern similar to that described for 56 week treatment (Doan et al., 2013)

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