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Cultures were left to express protein for 1620 h

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The fate of serine metabolism on macrophage polarization was investigated and a novel mechanism was proposed by which serine deficiency stimulates the M1 phenotype and inhibits STAT6-mediated M2 polarization by activating p38-dependent JAKSTAT-1 axis [286], suggesting that serine metabolism may be an underlying target

Additionally, loss of -syn has been associated with an increase in dopaminergic release (Senior et al., 2008