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PKA may potentially phosphorylate various target proteins, possibly initiating cellular responses

The effect of N-acetylcysteine on biofilms: Implications for the treatment of respiratory tract infections

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64 Findings from H9N2 swine influenza virus-infected murine model indicated that N-acetylcysteine ameliorated pulmonary edema and inflammatory response, reduced myeloperoxidase activity in the lungs, decreased total and differential cell counts, neutrophils, macrophages, IL-6, IL-1, TNF-, and CXCL-10 in the bronchoalveolar lavage fluid, and inhibited protein expression of TLR4 and its mRNA in the lungs