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SKU: 6562624011
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Radi, R., Rodriguez, M., Castro, L

Brzezinski A

Thabtah, F., Kamalov, F

The prevailing hypothesis points to a tissue-specific immune mechanism, where spinal entheseal IL-17 production may be IL-23-independent, or where IL-23 is critical for disease initiation but not its perpetuation[27, 34]